15/10/2015 BEQ

15/10/2015 BEQ

1 CONSANGUINEOUS MARRIAGE ---------------DR. ANIL

2 NOURISHMENT--------------------------------------DR RAJANI

3 UNIDIGITAL CLUBBING--------------------------DR.GOKUL

4 FUNNEL CHEST-------------------------------------DR WINNI

08/10/2015 BEQ

08/10/2015 BEQ

1 HICCUPS CAUSES --------------------------------------DR WINNI

2 TYPES OF FEVER---------------------------------------DR AMAR

3 DIRECT PERCUSSION OVER CLAVICLE---------DR YUNUS

4 SPLEEN SURFACE MARKING  ---------------------DR SRINIVAS

5 AUSCULTO PERCUSSION-------------------------------------------DR ANEESH

01/10/2015 BEQ

01/10/2015 BEQ

1 tripod position in asthma -------------------- DR AMAR

2. Massive hemoptysis -------------------------DR YUNUS

3. COMPLICATIONS OF COUGH----DR ANEESH

4.. PAO2 NORMAL VALUES   ---------DR SRINIVAS

5. SMOKING INDEX      ----------------DR ANIL

6. dry drowning and wet drowning--------DR RAJANI

7. division between upper lower respiratory tract  ------DR GOKUL

WelCome !!!! Anuroop & Umesh !!!

WelCome !!!! Anuroop & Umesh !!!

FORMOTEROL

BEQ 23/9/15


1 BULLAE ,EMPHYSEMA differences-                                       Dr amar

2 MECHANISM OF HEMOPTYSIS IN FUNGAL BALL                Dr yunus

3. air crescent sign                                                                      Dr aneesh

4.FORMOTEROL - HALF LIFE, MECHANISM OF ACTION                                Dr srinivas

5 commenest fungal organisms in fungal ball                          Dr anil
                                                       
6 mechanysm of syncop in pah                                                  Dr rajani

7 cough syncope mechanism                                                     Dr gokul

8 built and nutrition    def                                                                  Dr winni  




ANS-BEQ 23/9/15

FORMOTEROL - HALF LIFE, MECHANISM OF ACTION 
half-life is 10 h.
Inhaled formoterol works like other β₂ agonists, causing bronchodilation by relaxing the smooth muscle in the airway so as to treat the exacerbation of asthma. The long duration of formoterol action occurs because the formoterol molecules initially diffuse into the plasma membrane of the lung cells, and then are slowly released back outside, where they can come into contact with β₂ adrenergic receptors. Formoterol has been demonstrated to have a faster onset of action than salmeterol as a result of lower lipophilicity, and has also been demonstrated to be more potent – a 12 µg dose of formoterol has been demonstrated to be equivalent to a 50 µg dose of salmeterol.